16 Reasons Why Chelorex Is Your Best Choice

16 Reasons Why Chelorex Is Your Best Choice

Chelorex 1)  Chelorex is formulated specifically for chronic metal poisoning (CMP) rather than acute metal poisoning. Synthetic chelation agents like DMPS, DMSA and EDTA were formulated for treatment of acute metal poisoning (AMP) which differs in many important respects from CMP.

2)  Chronic metal poisoning is characterized by depletion of glutathione, oxidative stress, impairment of chemical detoxification mechanisms and impaired thyroid function. Chelorex contains no synthetic chelating agents, therefore, causes no dangerous adverse reactions in chemically sensitive people (30 - 40% of persons receiving synthetic chelators have moderate to severe adverse responses.)

3)  Chelorex contains natural components which raise glutathione levels, reduce oxidative stress, preserve mitochondrial function and help to improve thyroid function.

4)  Chelorex helps to raise metallothionein which also enhances toxic metal excretion.

5)  Selenium, zinc and magnesium reduce toxicity of heavy metals and protect against trace mineral depletion.

6)  Provides safe and effective mobilization of toxic metals by enhancing the natural mechanisms for excretion through the biliary tract. While avoiding the toxic overload associated with synthetic chelators.

7)  Multiple potent antioxidants protect against free radical damage.

8)  Marked enhancement of GI excretion of toxic metals (1280% at 14 days) with protection against re-absorption of toxic metals from GI tract.

9)  Kidney damage is prevented because unlike EDTA, excretion is primarily intestinal.

10)  Effective for entire spectrum of toxic metals: cadmium, tin, lead, mercury, aluminum, nickel, antimony, arsenic, silver, beryllium, platinum, tungsten, thallium and uranium. This is important because the presence of multiple toxic metals significantly lowers the toxic threshold for each metal.

11)  Most cost effective of all chelators.

12)  Chelorex is the only oral chelating agent with actual clinical studies showing efficacy in reducing toxic metal loads.

13) Can be used safely for long-term protection for persons who must sustain chronic environmental exposure.

14)  Contains both water-soluble agents and lipid soluble agents, capable of penetrating the blood-brain barrier and the cell membrane so that CNS and intracellular metals can be mobilized.

15)  Safely lowers mercury levels in persons with amalgam fillings. Can be used to protect persons before, during and after amalgam replacement.

16)  No EDTA avoids risk of neurotoxic complex with mercury reported by *Duhr, et al

References:

Duhr EF, Pendergrass JC, Slevin JT & Haley BE. HgEDTA complex inhibits GTP interactions with the E-site of brain beta-tubulin. Toxicol Appl Pharmacol 122(2):273-280(1993)

ABSTRACT:

"We have found that EDTA and EGTA complexes of Hg2+, which conventional wisdom has assumed are biologically inert, are potentially injurious to the neuronal cytoskeleton. Tubulin, a major protein component of the neuronal cytoskeleton, is the target of multiple toxicants, including many heavy metal ions. Among the mercurials, inorganic mercuric ion (Hg2+) is one of the most potent inhibitors of microtubule polymerization both in vivo and in vitro. In contrast to other heavy metals, the capacity of Hg2+ to inhibit microtubule polymerization or disrupt formed microtubules cannot be prevented by the addition of EDTA and EGTA, both of which bind Hg2+ with very high affinity. To the contrary, the addition of these two chelating agents potentiates Hg2+ inhibition of tubulin polymerization. Results herein show that HgEDTA and HgEGTA inhibit tubulin polymerization by disrupting the interaction of GTP with the E-site of brain beta-tubulin, an obligatory step in the polymerization of tubulin.

Both HgEDTA and HgEGTA, but not free Hg2+, prevented binding of [32P]8N3GTP, a photoaffinity nucleotide analog of GTP, to the E-site and displaced bound [32P]8N3GTP at low micromolar concentrations. This complete inhibition of photoinsertion into the E-site occurred in a concentration- and time-dependent fashion and was specific for Hg2+ complexes of EDTA and EGTA, among the chelating agents tested. Given the ubiquity of Hg2+ in the environment and the widespread use of EDTA in foodstuffs and medicine, these mercury complexes may pose a potentially serious threat to human health and play a role in diseases of the neuronal cytoskeleton."


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These statements have not been evaluated by the food and drug administration.  The preceding information and/or products are for educational purposes only and are not meant to diagnose, prescribe, or treat illness. Please consult your doctor before making any changes or before starting ANY exercise or nutritional supplement program or before using this information or any product during pregnancy or if you have a serious medical condition.

 


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